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dc.contributor.authorGrunblatt, Eli
dc.date.accessioned2018-11-05T21:33:20Z
dc.date.available2018-11-05T21:33:20Z
dc.date.issued2014-05
dc.identifier.urihttps://hdl.handle.net/20.500.12202/4075
dc.identifier.urihttps://yulib002.mc.yu.edu/login?url=https://repository.yu.edu/handle/20.500.12202/4075
dc.descriptionThe file is restricted for YU community access only.
dc.description.abstractBreast cancer is one of the most well-known and widespread forms of cancer in the United States today with over 40,000 deaths being attributed to it annually. According to the American Cancer Society, over 90% of these deaths are due to the cancer spreading to other areas of the body.1 Previous studies have shown that this aggressive action by the cancer is due to specific types of breast cancer cells that are also known to be resistant to conventional treatments.2 In this project, I elucidate a new potential drug target found in these cells. If drugs against this target come into use in the clinic, it may result in prevention of breast cancer spreading and increased long term survival for patients.en_US
dc.description.sponsorshipJay and Jeanie Schottenstein Honors Programen_US
dc.language.isoen_USen_US
dc.publisherYeshiva Collegeen_US
dc.rightsAttribution-NonCommercial-NoDerivs 3.0 United States*
dc.rights.urihttp://creativecommons.org/licenses/by-nc-nd/3.0/us/*
dc.subjectBreast --Cancer --Genetic aspects.en_US
dc.subjectBreast --Cancer --Chemotherapy --Complications.en_US
dc.subjectCancer cells.en_US
dc.subjectDNA repair.en_US
dc.titleInvestigating the Role of the Anti-Apoptotic Protein ARC in Breast Cancer Cell DNA Repairen_US
dc.typeThesisen_US


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Attribution-NonCommercial-NoDerivs 3.0 United States
Except where otherwise noted, this item's license is described as Attribution-NonCommercial-NoDerivs 3.0 United States